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that is certainly, stimulation of CD74 with exogenous MIF may well produce an abnormal inflammatory state, as MIF is capable of stimulating CXCR2 and CXCR4 receptors in addition to CD74. CXCR2 and CXCR4 receptors when activated promote inflow of neutrophils and lymphocytes, respectively (63, 64). In distinction, activation of the pro-survival kina